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Conceivably, depolarizations caused by blocking KCNQ channels may reduce the ligand binding affinity of presynaptic MOR1 directly at the presynaptic terminal, or by promoting repetitive depolarizations in intrinsic burster neurons (Ramirez et al., 2011, 2012), sufficiently to decrease MOR1 signaling
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Established tendinopathy actually shows INCREASED (but abnormal) blood vessel growth accompanied by sensory nerve ingrowth — this pathological neovascularization contributes to pain
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