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Frontiers | The dysregulation of immune cells induced by uric acid: mechanisms of inflammation associated with hyperuricemia and its complications
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In addition, CS exposure also protected β-cells against oxidation (H 2 O 2 ) - or lipitoxity (palmitic acid, PA)- or glucotoxity (high concentration glucose, HG)-induced cell death at concentrations of 2.5 μM or 5 μM, respectively (Supplementary Figures S3B–F)
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